Silicosis

It is a type of pneumoconiosis.

Of these deaths, 46,000 were due to silicosis, 24,000 due to asbestosis and 25,000 due to coal workers pneumoconiosis.

Silicosis is a form of occupational lung disease caused by inhalation of crystalline silica dust.

Most common pneumoconiosis are silicosis, coal workers’ pneumoconiosis (CWP), and asbestosis.

It may often be misdiagnosed as pulmonary edema (fluid in the lungs), pneumonia, or tuberculosis.

Silicosis increases the risk about 30-fold.

Progressive massive fibrosis (PMF), characterized by the development of large conglomerate masses of dense fibrosis (usually in the upper lung zones), can complicate silicosis and coal worker's pneumoconiosis.

The pneumatic hammer drill was introduced in 1897 and sandblasting was introduced in about 1904, both significantly contributing to the increased prevalence of silicosis.

Using a jackhammer to break up concrete pavement may expose the operator to hazardous dust containing respirable crystalline silica.

With retraction of the lung tissue, there is compensatory emphysema.

Silica dust and fiberglass dust exposure can also lead to COPD, with the risk unrelated to that for silicosis.

Characteristic lung tissue pathology in nodular silicosis consists of fibrotic nodules with concentric "onion-skinned" arrangement of collagen fibers, central hyalinization, and a cellular peripheral zone, with lightly birefringent particles seen under polarized light.

Birefringence of particles biopsied from pulmonary nodules indicates silicosis.

Silicosis is a form of occupational lung disease caused by inhalation of crystalline silica dust.

Inhaling finely divided crystalline silica is toxic and can lead to severe inflammation of the lung tissue, silicosis, bronchitis, lung cancer, and systemic autoimmune diseases, such as lupus and rheumatoid arthritis.

In the United States, a 1930 epidemic of silicosis due to the construction of the Hawk's Nest Tunnel near Gauley Bridge, West Virginia caused the death of at least 400 workers.

The Hawks Nest Tunnel disaster was a large-scale incident of occupational silicosis as the result of the construction of the Hawks Nest Tunnel near Gauley Bridge, West Virginia, as part of a hydroelectric project.

Crystalline silica of respirable size is a recognized human carcinogen and may lead to other diseases of the lungs such as silicosis and pulmonary fibrosis.

Silicon (Si) is the second most common element in the Earth's crust (oxygen is the most common).

Inhalation of crystalline silica dust may lead to silicosis, an occupational lung disease marked by inflammation and scarring in the form of nodular lesions in the upper lobes of the lungs.

Much of the thousands of tons of debris resulting from the collapse of the Twin Towers was pulverized concrete, which is known to cause silicosis upon inhalation.

Pneumonoultramicroscopicsilicovolcanoconiosis is a word coined by the president of the National Puzzlers' League as a synonym for the disease known as silicosis.

The pneumatic hammer drill was introduced in 1897 and sandblasting was introduced in about 1904, both significantly contributing to the increased prevalence of silicosis.

It tends to break up quickly, creating large quantities of dust, exposing the operator to the potential development of silicosis, a debilitating lung disease.

Inhalation of crystalline silica is harmful to the lungs, causing silicosis.

Also, the mining establishment of Delamar Ghost Town, Nevada was ruined by a dry-mining process that produced a silicosis-causing dust.

Miners breathing the dust often developed silicosis and the town became known as a "widow-maker."

Although silicosis has been known for centuries, the industrialization of mining has led to an increase in silicosis cases.

There are numerous occupational hazards associated with mining, including exposure to rockdust which can lead to diseases such as silicosis, asbestosis, and pneumoconiosis.

Silicosis is a form of occupational lung disease caused by inhalation of crystalline silica dust.

It is marked by inflammation and scarring in the form of nodular lesions in the upper lobes of the lungs.

Characteristic lung tissue pathology in nodular silicosis consists of fibrotic nodules with concentric "onion-skinned" arrangement of collagen fibers, central hyalinization, and a cellular peripheral zone, with lightly birefringent particles seen under polarized light. It is marked by inflammation and scarring in the form of nodular lesions in the upper lobes of the lungs.

It is marked by inflammation and scarring in the form of nodular lesions in the upper lobes of the lungs.

Silicosis (particularly the acute form) is characterized by shortness of breath, cough, fever, and cyanosis (bluish skin).

It may often be misdiagnosed as pulmonary edema (fluid in the lungs), pneumonia, or tuberculosis.